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BNP has important pathophysiological significance, it can promote sodium excretion, urination, has a strong vasodilating effect, can resist the vasoconstriction effect of renin-angiotensin-aldosterone system (RAAS), and is a major endocrine system in the human body to resist volume overload and hypertension like ANP. Cardiac dysfunction greatly activates the natriuretic peptide system, and increased ventricular load leads to BNP release.



Brain Natriuretic Peptide (BNP), also known as B-type Natriuretic Peptide, Brain Natriuretic Peptide, is another member of the natriuretic peptide system after cardiac natriuretic peptide (ANP), because it was first isolated from the pig brain by Japanese scholar Sudoh in 1988, in fact, it mainly comes from the ventricles.

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